New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo

AJL Ham; BP Engelward; H Koc; R Sangaiah; LB Meira; LD Samson; JA Swenberg

doi:10.1016/j.dnarep.2003.11.003

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New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo

AJL Ham, BP Engelward, H Koc, R Sangaiah, LB Meira, LD Samson and JA Swenberg

DNA REPAIR, Vol.3(3), pp.257-265

04/03/2004

DOI: https://doi.org/10.1016/j.dnarep.2003.11.003

Abstract

Science & Technology

Life Sciences & Biomedicine

Genetics & Heredity

Toxicology

GENETICS & HEREDITY

TOXICOLOGY

immunoaffinity-LC-MS/MS methodology

1

N-6-ethenodeoxyadenosine

lung

liver

alkyladenine DNA glycosylase

mice

NUCLEOTIDE EXCISION-REPAIR

OXIDATIVE STRESS MARKERS

ETHYL CARBAMATE URETHANE

VINYL-CHLORIDE

LIPID-PEROXIDATION

ESCHERICHIA-COLI

POTENTIAL ROLE

MASS-SPECTROMETRY

FORMING CHEMICALS

MOUSE CELLS

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Title: New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo
Creators: AJL Ham
BP Engelward
H Koc
R Sangaiah
LB Meira
LD Samson
JA Swenberg
Publication Details: DNA REPAIR, Vol.3(3), pp.257-265
Publisher: ELSEVIER SCIENCE BV
Date published: 04/03/2004
Date submitted: 17/05/2017
Identifiers: 99516744002346
Academic Unit: University of Surrey
Language: English
Resource Type: Journal article

New immunoaffinity-LC-MS/MS methodology reveals that Aag null mice are deficient in their ability to clear 1,N-6-etheno-deoxyadenosine DNA lesions from lung and liver in vivo

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