Human CD133(+) Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling

LS Barcelos; C Duplaa; N Kraenkel; G Graiani; G Invernici; R Katare; M Siragusa; M Meloni; I Campesi; M Monica; A Simm; P Campagnolo; G Mangialardi; L Stevanato; G Alessandri; C Emanueli; P Madeddu

doi:10.1161/CIRCRESAHA.108.192138

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Human CD133(+) Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling

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Human CD133(+) Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling

LS Barcelos, C Duplaa, N Kraenkel, G Graiani, G Invernici, R Katare, M Siragusa, M Meloni, I Campesi, M Monica, …

CIRCULATION RESEARCH, Vol.104(9), pp.1095-U199

08/05/2009

DOI: https://doi.org/10.1161/CIRCRESAHA.108.192138

Abstract

Science & Technology

Life Sciences & Biomedicine

Cardiac & Cardiovascular Systems

Hematology

Peripheral Vascular Disease

Cardiovascular System & Cardiology

CARDIAC & CARDIOVASCULAR SYSTEMS

HEMATOLOGY

PERIPHERAL VASCULAR DISEASE

ischemia

wound healing

diabetes

stem cells

angiogenesis

EMBRYONIC STEM-CELLS

GROWTH-FACTOR

IN-VITRO

ENDOTHELIAL-CELLS

FOOT ULCERS

MOUSE SKIN

PATHWAY

WOUNDS

NEOVASCULARIZATION

TRANSPLANTATION

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Title: Human CD133(+) Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling
Creators: LS Barcelos
C Duplaa
N Kraenkel
G Graiani
G Invernici
R Katare
M Siragusa
M Meloni
I Campesi
M Monica
A Simm
P Campagnolo
G Mangialardi
L Stevanato
G Alessandri
C Emanueli
P Madeddu
Publication Details: CIRCULATION RESEARCH, Vol.104(9), pp.1095-U199
Publisher: LIPPINCOTT WILLIAMS & WILKINS
Publication Date: 08/05/2009
Identifiers: 99511012002346; WOS:000265917500011
Academic Unit: University of Surrey
Language: English
Resource Type: Journal article

Human CD133(+) Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling

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