Response to: Depolarization vs. repolarization: what is the mechanism of ventricular arrhythmogenesis underlying sodium channel haploinsufficiency in mouse hearts?

Journal article

Peer reviewed

K. Jeevaratnam, L. Guzadhur, Y. M. Goh, A. A. Grace and C. L. -H. Huang

ACTA PHYSIOLOGICA, Vol.218(4), pp.236-238

12/2016

PMID: 27272698

Abstract

Life Sciences & Biomedicine

Physiology

Science & Technology

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Title: Response to: Depolarization vs. repolarization: what is the mechanism of ventricular arrhythmogenesis underlying sodium channel haploinsufficiency in mouse hearts?
Creators: K. Jeevaratnam - Perdana University
L. Guzadhur - University of Cambridge
Y. M. Goh - Universiti Putra Malaysia
A. A. Grace - University of Cambridge
C. L. -H. Huang - University of Cambridge
Publication Details: ACTA PHYSIOLOGICA, Vol.218(4), pp.236-238
Publisher: Wiley
Number of pages: 3
Publication Date: 12/2016
Identifiers: 991109885802346; WOS:000388141700005
Academic Unit: School of Veterinary Medicine
Language: English
Resource Type: Journal article