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Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice
Journal article   Peer reviewed

Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice

A Unnikrishnan, JJ Raffoul, HV Patel, TM Prychitko, N Anyangwe, LB Meira, EC Friedberg, DC Cabelof and AR Heydari
FREE RADICAL BIOLOGY AND MEDICINE, Vol.46(11), pp.1488-1499
01/06/2009
DOI: https://doi.org/10.1016/j.freeradbiomed.2009.02.021

Abstract

Science & Technology Life Sciences & Biomedicine Biochemistry & Molecular Biology Endocrinology & Metabolism BIOCHEMISTRY & MOLECULAR BIOLOGY ENDOCRINOLOGY & METABOLISM Apurinic/apyrimidinic endonuclease 1/redox factor-1 Redox activity Base excision repair Oxidative DNA damage NF-kappa B Apoptosis Liver Free radicals NF-KAPPA-B POLYMERASE-BETA HAPLOINSUFFICIENCY STRANDED DEOXYRIBONUCLEIC-ACID URACIL-DNA GLYCOSYLASE HUMAN-CELLS BINDING ACTIVITY DAMAGING AGENTS APURINIC SITES NITRIC-OXIDE ACTIVATION
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