Evidence that inward rectifier K(+) channels mediate relaxation by the PGI(2) receptor agonist cicaprost via a cyclic AMP-independent mechanism

NN Orie; CH Fry; LH Clapp

doi:10.1016/j.cardiores.2005.08.004

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Evidence that inward rectifier K(+) channels mediate relaxation by the PGI(2) receptor agonist cicaprost via a cyclic AMP-independent mechanism

NN Orie, CH Fry and LH Clapp

CARDIOVASCULAR RESEARCH, Vol.69(1), pp.107-115

01/01/2006

DOI: https://doi.org/10.1016/j.cardiores.2005.08.004

Abstract

Science & Technology

Life Sciences & Biomedicine

Cardiac & Cardiovascular Systems

Cardiovascular System & Cardiology

CARDIAC & CARDIOVASCULAR SYSTEMS

prostacyclin analogue

vascular smooth muscle

inward rectifier potassium channels

relaxation

EDHF

ARTERIAL SMOOTH-MUSCLE

GUINEA-PIG AORTA

POTASSIUM CHANNELS

NITRIC-OXIDE

PROSTANOID RECEPTORS

BLOOD-VESSELS

G-PROTEINS

RAT

PROSTACYCLIN

CORONARY

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Title: Evidence that inward rectifier K(+) channels mediate relaxation by the PGI(2) receptor agonist cicaprost via a cyclic AMP-independent mechanism
Creators: NN Orie
CH Fry
LH Clapp
Publication Details: CARDIOVASCULAR RESEARCH, Vol.69(1), pp.107-115
Publisher: OXFORD UNIV PRESS
Date published: 01/01/2006
Date submitted: 28/02/2012
Identifiers: 99512107902346
Academic Unit: University of Surrey
Language: English
Resource Type: Journal article

Evidence that inward rectifier K(+) channels mediate relaxation by the PGI(2) receptor agonist cicaprost via a cyclic AMP-independent mechanism

Abstract

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